By Rebecca M. Clarke
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Additional info for Antisocial behavior : causes, correlations and treatments
1, all statistically significant, but none significantly different from the respective ORs associated with ASPD. After adjustment for 26 Risë B. Goldstein and Bridget F. 7). 3). However, no OR for any specific disorder associated with AABS differed significantly from that associated with ASPD. , 2009; Mulder, Joyce, Frampton, Luty, & P. F. , 1997), clinical studies that have examined Axis II comorbidity in patients with mood and anxiety disorders have documented poorer outcomes of pharmacologic as well as psychosocial treatment associated with any PD, specific PD clusters, and various specific PDs in patients with mood and anxiety disorders.
5% but no respondent met current criteria. 3% on both lifetime and current bases for OCD. To our knowledge, general population prevalences of DSM-III GAD have not been reported. For the other anxiety disorders, however, as was the case with mood disorders, prevalences reported by Black, Baumgard, and Bell (1995) among men with lifetime DSM-III ASPD are substantially greater than those of DSM-III anxiety disorders observed in the ECA (cf. Eaton, Dryman, & Weissman, 1991; Helzer, L. N. , 1984; L.
The difference by antisocial syndrome was, once again, statistically nonsignificant, but prevalences in both antisocial groups were significantly greater than among respondents with no lifetime antisocial syndrome (Marmorstein, 2006). , 1994). , 2005). , 1994). 05; Marmorstein, 2006). 5 compared with respondents who had neither ASPD nor CD (Goodwin & Hamilton, 2003). , 2005). , 2006). 0%, which did not differ significantly but were both significantly greater than the lifetime prevalence among nonantisocial respondents.